Aspartame Dangers – this will shock you!

aspartame sodasAspartame Dangers will shock you!

Do you drink a diet coke or other diet sodas?  How many  sugar free yoghurts do you eat?   Read Aspartame Dangers and be shocked. What about aspartame flavoured cereal, chewing gum, puddings, cakes, juices or one of the other 6,000 products that contain aspartame?  Aspartame Dangers will go into how this chemical affects your body.  If you have time, take a look at this video:  (See Part 2 and Part 3 of this trilogy).

NutraSweet and others

Aspartame is commonly known as NutraSweet, Equal, Spoonful, Equal Measure etc.  You will be shocked at how this poison was passed initially by the FDA (Food and Drug Administration).  Now it is consumed by millions but will you keep using it after reading gumthis post?

But first, I want to relay to you how aspartame was passed as safe by the FDA. This was without proper research, with fraudulent and misleading studies by the then aspartame producing company G D Searle. As well as this, political misdemeanours abounded by Donald Rumsfeld, the CEO of Searle at the time of FDA approval.

How it started

The story starts way back in 1965, when a sweet substance was discovered, quite by accident, by James M. Schlatter, a chemist working for G D Searle & Company.  He was working on an anti ulcer drug. After some of the powder was spilt he licked his fingers to pick up a piece of paper and noticed a very strong sweet taste…

A long story

The story of aspartame is a long one, too long to cover in this article so I will try and give you the salient points. This will allow you to make an informed decision as to whether you wish to continue consuming it.

Dr John OlneyA good place to start

In the spring of 1971 a neuroscientist Dr John Olney was studying MSG (monosodium glutamate). He informed Searle that his studies had revealed that aspartic acid, one of the ingredients of aspartame, caused holes in the brains of infant mice.  (Dr Olney was responsible for having msg removed from baby foods).  One of Searle’s researchers, Ann Reynolds, confirms Olney’s findings in a similar study.

Controversial studies

In November 1971, Searle started a 115 week study (No. E-77/78) to test aspartame with 360 weanling albino rats, 190 of each sex.  They also did 2 other studies using mice and it was these particular studies that caused much controversy.

FDA approved aspartame

The FDA approved aspartame in July 1974 for limited use as a sugar substitute for sweetening hot beverages, cereals, gum and dry bases. It was not approved for baking goods, cooking or carbonated beverages.  FDA scientists found serious flaws in 13 tests related to genetic damage which was submitted by Searle.  So why did they give their approval?

A formal objectionJames Turner

in August 1974, a formal objection was filed.  Dr John Olney, James Turner and Label Inc. (legal action for buyers’ education and labeling) expressed serious concerns  They stated that they believed that aspartame could cause brain damage.  Aspartame’s effects on children was of particular concern to them.

Concerns over other drug studies

We’re only talking about aspartame here. But in July 1975 the FDA commissioner Dr Alexander Schmidt appointed a Task Force to look into 25 studies for the drugs Flagyl, Aldactone, Norpace and Aspartame.  This was because of concerns of Searle’s responses to queries about the testing of their drug Flagyl. Also there were serious and unexpected side effects from other drugs Searle developed.   The Olney studies and information, started controversy within the FDA as to the validity of Searle’s research of aspartame and other drugs.  All of the studies were either by Searle or done for Searle, ie. Hazleton Laboratories.  Eleven of the studies involved aspartame.

 put on hold

In December 1975 there was a hold of approval by the FDA for aspartame, due to findings of the Task Force.  The evidence of the aspartame pivotal studies were protected under FDA seal on December 3rd 1975.

Searle building an aspartame facility

Meanwhile, Searle was building a production facility for aspartame and had so far spent 19.7 million dollars.   On 8th December 1975, stockholders filed a class action lawsuit against Searle. They accused them of concealing information regarding the quality of research at Searle. This being in violation of the Securities and Exchange Act.

500 page report completed.

In March 1976 a 500 page report by the FDA Task Force was completed.  Here are some of the conclusions from this report. Searle’s research practices have been taken from Mark D. Gold’s FDA Dockets Submittal which has all relevant references therein.

“We have noted that Searle has not submitted all the facts of experiments to the FDA, retaining unto itself the unpermitted option of filtering, interpreting and not submitting information which we would consider material to the safety evalution of the product….Finally we have found instance of irrelevant or unproductive animal research where experiments have been poorly conceived, carelessly executed, or inaccurately analyzed or reported.”


“Some of our findings suggest an attitude of disregard for FDA’s mission of protection of the public health by selectively reporting the results of studies in a manner which allay the concerns of questions of an FDA reviewer.”

Worrying research practices

“Excising masses (tumors) from live animals, in some cases without histologic examination of the masses, in others without reporting them to the FDA.”  When Searle’s representatives were questioned they stated “these masses were in the head and neck areas and prevented the animals from feeding.”

“Failure to report to the FDA all internal tumors present in the experimental rats, e.g., polyps in the uterus, ovary neoplasms as well as other lesions.”

“Instead of performing autopsies on rhesus monkeys that suffered seizures after being fed aspartame, the company had financed a new monkey seizure study with a different methology that showed no problems.”

Animals which had died were sometimes recorded as being alive and vica versa. “These include approximately 20 instances of animals reported as dead and then reported as having vital signs normal again at subsequent observation periods.

“Never seen anything as bad as Searle’s studies!”
“even with some fraud thrown in, it didn’t work”.

Phillip Brodsky described the 1975 FDA Task Force members as some of the most experienced drug investigators.  Brodsky was the lead investigator. He went on to state that he had never seen anything as bad as Searle’s studies.

The FDA Commissioner at the time, Alexander Schmidt stated “[Searle’s studies were] incredibly sloppy science.  What we discovered was reprehensible.”

Senator Kennedy says “profoundly disturbing.”

Senator Edward Kennedy at the April 1976 hearings, before the Senate subcommittee on Labor and Public Welfare stated: “The extensive nature of the almost unbelievable range of abuses discovered by the FDA on several major Searle products is profoundly disturbing.

research into aspartameAn independent study

To finish Part One of this post, take a look at this research. This was done by an independent researcher who just wanted to know the truth. Victoria Inness-Brown started the study because of concerns for her family who drank many diet sodas.

Victoria came across ‘The Bressler Report’ which we will discuss in Part 2. Hence, she decided to do her own experiment so she could see for herself if aspartame did actually produce tumours.  She used rats for the study, 108 of them that she raised herself.  Victoria had a control group as well as an experimental group.

NutraSweet used in study

male rat tumour aspartameShe could not obtain the pure form of aspartame because she wasn’t a food manufacturer consquently she used NutraSweet which she put into the rats’ water.  The rats were given the acceptable daily limit or acceptable daily intake (ADI). This is set by the FDA which is 50mg per kilogram.  The research took 2.5 years, ie. 30 months to complete.  Also, she had no help with finance but stated that it wasn’t too expensive to do the study, just time consuming.  She did all the care for the animals and was meticulous with recording her research.

As you can see by the images here, the results of the study are quite horrendous.  If you wish to look at more information regarding this study you can go to her page at or check out the narrative of her special interview with Victoria Inness-Brown and Dr Mercola.

Ches PowerIn Part 2 of this article, the Bressler Report is instigated by the FDA and Donald Rumsfeld joins G D Searle. President Ronald Reagan comes to power in 1981. Approval of aspartame comes soon after.

H. Pylori Infection – are you at risk?

Robin Warren
Dr Robin Warren

I know what you’re saying – what on earth is H. pylori infection and have I got it?  I want to tell you a story of two amazing researchers who discovered why we get stomach ulcers and even proved it.

Trouble was, nobody would take any notice of them.  Why?  Because the powers that be and those that vetted all new research and cures, ignored their work. It went against everything previously taught. It was pure dogma and closed-mindedness.  H. pylori stands for Helicobacter pylori a then unknown bacteria.

Dr Robin Warren

So who am I talking about?  I’m speaking initially of Dr Robin Warren, a pathologist at the Royal Perth Hospital (RPH) in W. Australia.  In 1979, he found evidence of bacteria in a stomach biopsy from a man with severe gastritis.  Dr Warren was rather curious about this strange finding and a pattern began to show.  Whenever there was chronic gastritis, the bacteria was present.  He also noticed that the more inflammed the stomach lining was, the more abundant the bacteria.

Dr Barry Marshall

Dr Marshall
Dr Barry Marshall

In 1981, Barry Marshall, who was in his second year of internal medicine, joined Dr Warren, in his investigation into the gastric spiral bacteria.  Marshall’s interest was soon aroused. One of the patients was a woman he had seen on his ward. She had severe stomach pain but no diagnosis.  She had been referred to a psychiatrist. Her prescription was  antidepressents, for want of a better treatment.  But she had some redness in the stomach. With a biopsy,  Warren’s bacteria was found in her stomach.

No pre-conceived ideas

Warren had approached other colleagues to join him. But Marshall’s obvious advantage was that he had no pre-conceived ideas about the possibility of bacteria living in the acid-filled stomach.  He did not come from a background of gastroenterology. His knowledge to that date was based on science rather than dogma which seems to be a requirement to learn specialist medicine.

Attempts at culturing H. pylori

In April of 1982, Warren and Marshall had attempted to culture samples from 30 patients but without success.  During the Easter holidays, the hospital was fighting an outbreak of Staphylococcus aureus. Consequently, the latest culture attempt was left in the uncubator over the Helicobacter pylori cultureholidays.  It was normal procedure to leave the cultures to incubate for just two days, after which, if no growth was evident the cultures were discarded.  When the staff came back after the holiday, they were surprised to find that the cultures were showing growth.  It was then apparent that this spiral bacteria was a slow grower.  For the previous 6 months, lab technicians were throwing away viable cultures after 2 days. This is the normal amount of time a culture is left to grow.  It just shows how paradigms and habits can sometimes keep research blinkered.

Their research rejected

In July 1982, Marshall moved to Port Hedland Hospital, a position allocated to him within his training.  Marshall used this time to do an extensive literature search and write up all the work they had done thus far ready for presentation.  Warren and Marshall presented their findings to the College of Physicians at RPH in October 1982. Their work was not very well received.

Skepticism ruled

There was much skepticism and many objections one of which was the link between gastritis and duodenal ulcers.  It was common knowledge that gastritis was associated with gastric ulcers but not duodenal ulcers.  Marshall did some more research and found that there had already been evidence way back in the 1950’s.  The Mayo Clinic  carried out post- mortems on vehicle fatalities or partial gastrectomy patients. They were h. pylori infectionlooking for the evidence of gastritis and ulcers.  Those who had duodenal ulcers (N = 250) were all found to also have gastritis. This evidence way back in the 1950’s supported Warren and Marshall’s study. But still, the ulcer research fraternity ignored this relationship between gastritis and duodenal ulcers.  Marshall speculated that their work was dismissed because it did not fit in with current beliefs and paradigms.

Marshall goes to Fremantle Hospital

He was then offered a position at Fremantle Hospital as a senior registrar with funding to continue the research.  The smallest of teaching hospitals in Perth, Fremantle had a tradition of experimentation and open-mindedness and Marshall took up the offer.  The only downside was that he was forced to finish his collaboration with Warren.  Even so, Marshall was surrounded by an enthusiastic group of helpers at Fremantle. One of which was the pathologist Ross Glancy who joined the team.

The Bismuth Test

H. pylory infection
A clear zone of inhibition with bismuth

It was here that Marshall found out why ‘colloidal bismuth subcitrate’ (CBS) reduced the relapse rate of ulcers.  He summised that CBS should have anti-microbial properties and he proceeded to test it.  He soaked a filter paper disc in CBS and placed it in the middle of a Petri dish inoculated with H. pylori.  After a few days and to his delight, there was a clear zone of inhibition around the paper disc.

In 1983 a Dr Martin Skirrow arranged for a presentation to be made to the European Campylobacter Meeting in September 1983.  Marshall visited Dr Martin Skirrow in Worcester England.  Dr Skirrow’s registrar successfully isolated the bacteria 3 days later.

Proof that organism was worldwide

This proved that the spiral organism was in fact worldwide and not just a product of Australia.  Further proof came from several groups around the world who were also obtaining similar results.  The European group came about from the campylobacter group Marshall had met in Brussels in 1983 and they were now a close knit community sharing and writing remarkable accounts of the research. This was to change the lives of millions of gastritis and ulcer sufferers.

Research still rejected!

1984 was a difficult year for Marshall.  Most of his work was rejected for publication and the research was met with constant criticism.  Marshall was disbelieved and the work was disputed, mainly on the premise that it simply could not possibly be true.  Everyone knew nothing could live in the acid stomach. After all, it was written in the text books!  This was despite the fact that patients were being treated and cured with a disease that many had suffered for years.  For the treatment to be recognised and accepted within the medical community, Marshall had to come up with something radical.


h. pylori infectionOut of desperation and frustration, he came up with the idea of infecting himself.  His research had shown that H. pylori was sensitive to antibiotics. Marshall himself had successfully treated gastritis patients using antibiotics and bismuth.  He isolated the culture he was to drink, from a patient with active gastritis. There was also an antibiotic available, metronidazole,  which  was effective in irradicating the cultured bacteria. The patient was then given a 2 week treatment of bismuth and metronidazole and was cured of the disease.


Marshall took Tagamet for one month

Marshall took an anti-acid drug known as cimetidine (Tagamet) for one month* before drinking the  3 day culture.  Seven days later he became very ill and began to vomit.   An endoscopic biopsy of his stomach showed, much to his relief and obvious satisfaction, that he indeed had gastritis with the presence of Helicobater pylori.  Marshall began antibiotic treatment a few days later and his symptoms soon disappeared.  Another endoscopy proved all traces of Helicobater pylori had gone. This was irrefutable evidence linking Helicobater pylori infection of a normal stomach to gastric inflammation.

Marshall returns to the Royal Perth

At the end of 1984 Marshall moved back to Royal Perth Hospital to conduct a double blind trial for antibiotics to see if they could cure duodenal ulcers.  The Australian Medical Research Council was to fund the trail on the condition Marshall could enlist a large number of patients.  He would be working with Robin Warren again along with other colleagues.  He and Warren still kept in touch and discussed the papers they were writing for The Lancet.  They would often meet for dinner with their wives.  Marshall was anxious to share the news about his self experimentation and Warren was equally excited.  Now they had first hand confirmation of infection and documented results.

Ignorant reporter!

At 5 am the next morning, Warren received a phone call from the USA.  It was an inconsiderate journalist.  He asked the usual stupid questions about the bacteria being just a harmless commensal and not a pathogen at all.

Warren loses his temper

Warren irritated, blurted out that they knew it was a pathogen because Barry Marshall had just infected himself and ‘damn near died’.  The unreleased work was now in the hands of a journalist from the ‘Star’ tabloid newspaper.  Freakish, bizarre and somewhat far-fetched copy was their speciality and this story was just up their street.  The next day the ‘Star’ published “Guinea-pig doctor discovers new cure for ulcers…and the cause”.

Warren & Marshall
Dr Barry Marshall and Dr Robin Warren 1984

As it happens, this unwanted publicity was to change Marshall’s life for the good and it was thanks to Warren’s temper.  A continuous stream of patients from the USA had read the story and were desperate for the new treatment.  Marshall was able to help these patients by proxy in the USA as early as 1984.  This documented story and the subsequent records from the treated patients was also important.  Another medic tried to claim credit for being the first discoverer.  Marshall was able to prove his claim to be the first along with Warren.

Dogma kept patients in misery for years.

It wasn’t until 1994 and after many years of research that the medical community started to come around to the theory of bacterial infection and the consequence of ulcers. The medical profession had thought that peptic ulcer disease was caused by too much stomach acid. This fact was laid down in medical textbooks.  Because of dogma, many long suffering patients tolerated untold misery and unpleasant surgery.  Once a patient had an ulcer it was difficult to erradicate. Most kept their ulcers for life, controlling their flare-ups with drugs.

Intransigent medical community capitulate

Marshall and Warren
Prof Barry Marshall and Dr Robin Warren

How lucky it was that these two researchers were as tenacious as they were. One driven to putting his own life at risk to prove something that had already been proven.  But despite this proof, it was not recognised or accepted by the medical fraternity.  Unnecessary years of suffering had to be endured because of the pig-headedness of the medical community.  Warren first was suspicious of this bacteria way back in 1979.  If only the medical community had taken notice and started listening and upping the research.  Then all those years of  suffering and misery and death for so many patients, could have been avoided.

Marshall and Warren’s nobel prize speeches

In Barry Marshall’s nobel prize speech he mentioned that Warren’s wife had indicated in 1982 that there may be a nobel prize in the offing for this research.  Marshall cynically replied that if so, it wouldn’t happen for ages, perhaps 1986.  Dr Robin Warren and Dr Barry Marshall were jointly awarded the Nobel Prize for their research in 2005!

26 years of unnecessary suffering and death!

From 1979 – 2005. For 26 years patients were suffering, being operated on and dying needlessly, all because of medical dogma   If anyone’s interested here’s a link to their nobel prize speeches:

h. pylori infection - causesWhy still PPIs? – pharma profits?- you bet!

Out of curiosity, I had a little look around for the now known treatment of gastritis and/or ulcers.  We already know that Marshall was treating and curing patients with ulcers way back in 1984.  As Marshall himself writes: “I was successfully experimentally treating patients who had suffered with life threatening ulcer disease for years. Some of my patients had postponed surgery which became unnecessary after a simple 2 week course of antibiotics and bismuth”.

So why is it that patients with ulcers now, are still being treated with proton pump inhibitors such as Nexium.  Why is the medical profession not using just antibiotics and bismuth as Marshall did way back in 1984?  Could it be something to do with still pushing cimetidine and other money spinning PPI drugs?

* Marshall took an anti-acid drug known as cimetidine (Tagamet), a proton pump type inhibitor, for one month before drinking the H. pylori culture. Why? To stop his stomach from producing acid which would allow the bacteria to take hold.  This proves that taking these drugs seriously impairs your capacity to digest and absorb nutrients because of induced low stomach acidity. It also suggests that low stomach acid, not excess acid, is the main Ches Power Authorcause of digestive problems, which can lead to ulcers and cancer.  See my previous post at



Proton Pump Inhibitors Side Effects

Proton pump inhibitors side effectsProton Pump Inhibitors Side Effects. Do you suffer from indigestion, heartburn or other digestive problems.  Perhaps you’ve been diagnosed with GERD (Gastroesophageal Reflux Disease).  This used to be called heartburn but now it’s been allocated a fancy name and given the class of a ‘disease’.

It is a condition we’ve brought upon ourselves. Problem is, it’s often being wrongly diagnosed which leads to inappropriate medication which can be detrimental to your health!

Now an ‘over the counter’ drug

If you have been prescribed with a PPI (proton pump inhibitor) drug, no doubt it will be something like Nexium (esomeprazole). Or perhaps Prilosec (omeprazole), Protonix (pantoprazole), Prevacid (lansoprazole) or Zegerid (omeprazole) for example.  But now these drugs can be bought without a prescription, known as an over the counter drug.  This means you can buy them willy nilly. This allows you to poison yourself even quicker than if you’d gone to the doctor for some.

Nexium BabyFor baby too, from 1 month old!

The problem is, these drugs should only be used for a maximum of 2 weeks (if not at all), otherwise the side effects can be profound.  Without the control of a prescription you are more likely to overtreat yourself with these drugs. Over use will likely cause  permanent symptoms of heartburn, acid regurgitation and dyspepsia after stopping the drug. This is despite you being in good health before starting the medication.  This is according to a study of more than 40% of healthy volunteers. They had never had symptoms of the above digestive problems but succumbed to the side effects of PPIs after cessation of treatment.  What’s more, you can now get this drug for use with babies from 1 month old.  I can see many parents trying this drug out to see if it stops baby crying without consulting a medic!

HeartburnToo much acid? No, too little!

So for those who consult their doctor about their heartburn or indigestion, what’s the diagnosis. Well, you will probably be been told that you have too much acid in your stomach. Or perhaps your acid is too strong and this is the cause of your problem?  If so, you are just 1 in 100 that actually do suffer from too much acid. This is because too much acid is quite unusual.  The majority of us who have heartburn, reflux or other indigestion symptoms are suffering from too little acid in our stomach, despite being mis-diagnosed the opposite.

Some diseases associated with low HCl and Mg deficiency

The older population as well as those with arthritis, osteoporosis, asthma, diabetes, depression, gallbladder disease are often lacking in hydrochloric acid.  Also, all the above conditions are associated with Mg deficiency.

Acid and AlkalineProblems caused by low acidity.

Weakened or inefficient stomach acid can lead to many problems, not least Mg deficiency. This can be a vicious circle because when Mg is deficient, stomach acid production is compromised which makes Mg absorption even more difficult.  The digestive process is vital for chemically changing our mineral intake into an absorbable form ie. ionization.  When a mineral complex such as for instance Mg citrate reaches the stomach, it needs the acidic conditions that the stomach produces, to break apart the constituent parts of the complex.  This changes Mg into its ionic form which can then be used by the body.

Apart from this, the lower esophageal sphincter (LES) which is at the entrance to the stomach, normally opens to allow food into the stomach. Once enough acid is in the stomach to process the food, the LES closes tightly.  The digestive process then progresses.  If the acidity within the stomach is too low, the LES signal to close can be compromised, allowing the weaker acid back into the esophagus, causing heartburn.

junk foodWhy do we do it?

Our digestion is often compromised by the junk food and drink that we consume.  After eating and drinking rubbish to excess, the first thing we turn to are antacids.  These are one of the most popular over the counter drugs and using them will do nothing for us except give temporary relief for a problem that we inflict on ourselves with our dysfunctional eating and drinking habits.  Our digestive systems have to try and deal with the sugary, greasy, non nutrional fast foods that we insist on burdening our bodies with.

How do PPIs (proton pump inhibitors) work?

Proton pump inhibitors, actually stop the acid producing pumps in the stomach and this can be positively harmful.  Normal stomach acid (hydrochloric acid or HCl) is stronger than battery acid.  It has this strength for a reason.  Neutralizing the normal stomach acid, makes it impossible for our bodies to absorb vitamins and minerals or digest our food efficiently.Lower acid with age

What reducing acid does

Reducing the stomach acid also compromises your defences against stomach infections and food poisoning.  Pathogens can get hold if not destroyed by the acidity.  Some bacteria such as Candida albicans and more seriously, H. pylori (Helicobactor pylori) can migrate from the intestines where they normally live without causing problems.  The reduced acidity will allow them to flourish within the stomach.  H. pylori has been proven to actually cause ulcers and if not eradicated can progress to induce stomach cancer.

These drugs, while giving temporary relief to sufferers, by slamming on the brakes of the acid-producing pumps, play havoc with these essential producers of acid.  When you stop taking the drug, acid is produced with a vengeance causing “rebound acid hypersecretion”. This rebound or withdrawal symptom is so intolerable that most patients go straight back onto taking the PPI again.  In other words they become reliant on the drug.  Never go ‘cold turkey’ with these drugs.  You have to withdraw from them very slowly to have any chance of getting off them for good.

Tums antacidAntacids

I would like to give a warning to those of you that take the antacid known as ‘Tums’. Antacids such as Tums actually advertise that they aid against osteoporosis.  This is despite antacids exacerbating osteoporosis and other calcifying type diseases.  Antacids that have a large quantity of Ca in them (mostly Calcium Carbonate CaCO3 which is in fact just chalk) will only make matters worse.  They will overload the system with Ca, subsequently this overload will not be controlled properly.  The excess Ca will migrate around the body, calcifying in places where it shouldn’t be.

Tums calcium contentAs I am always advocating, your Mg:Ca ratio should be as near to 1:1 as possible.  Despite advertising that they are ‘osteoporosis friendly’ these antacids are positively harmful.  The label information states “Each tablet contains: elemental calcium 200mg.” and on the same label: “Do not take more than 15 tablets in 24 hours.”  So, if you take the maximum dose of 15 tablets in a day, you will be consuming 3000mg of Ca.  This is a stupid amount of Ca to take which will exacerbate any calcification problemsTums tablets per day that you have and if you haven’t got them yet, you soon will! Taking these antacids will actually produce a magnesium deficiency by default. There is little chance of you upping your magnesium levels to match your calcium intake if you use antacids such as these.


Another worry is the advertisement image which shows what looks like a bag of sweets.  “Soft & Delicious” they advertise, “Chewy Delights”, and “Very Cherry”.  I can imagine many youngsters thinking these are sweets and most adults summizing that 15 tablets a day means they can’t be harmful.  Wrong.  This overdose of Ca is very harmful and I would suggest that you use real caution if you wish to consume these “Soft Delicious Chewy Delights”.  Beware!calcium overload

So, if you have too little acid in your stomach, why are you being prescribed a drug which actually turns off or reduces the production of acid?  What makes us think that from now on, our bodies can do without producing the correct concentration of acid to digest our food.  Why do we think that it will be healthy to stop the nutrients, vitimins and minerals from what we consume, being absorbed by way of our stomach acid.  How is it we’re daft enough to think we can get along without our stomach acid. How can our bodies protect us from toxins and bacterial overgrowth such as H. pylori and Candida albicans without the correct acidity to do the job?

Ironic isn’t it!

So here we have it.  You go to the doctor because of heartburn and other digestive symptoms.  You are diagnosed with having too much acid in your stomach.  You’re put on a drug which reduces the ‘too much acid’.  The PPI drug stops the production of stomach acid.  You think you’re cured and come off the drug.

Rebound acid hypersecretiion

Then ‘rebound acid hypersecretion’ or the build-up of acid that has not been released, is then unleashed with a vengeance.  What an irony.  You have too little acid and are diagnosed with too much acid, then the drug you take reduces your acid further. You stop using the drug, then the acid that has been suppressed is released. This gives you too much acid which you didn’t have in the first place! Now you’re on a spiral of having to take the drug to stop the rebound acid hypersecretion. Don’t do it!

First Do No Harm

kindnessWhy does this happen?  Because we have faith in those put into a position of trust such as medical professionals and supposedly moral drug manufacturers who we, the public, trust will ‘first do no harm‘.  We trust our doctors to know exactly what they are prescribing and that what they do prescribe is in our best interests and will not make our condition worse, and/or make us reliant on that drug, a drug such as Nexium.  Those of you who are taking Nexium and are trying to come off it, will know exactly what I mean.

Jonathan Wright, MD, states, “In 24 years of nutritionally oriented practice, I’ve worked with thousands of individuals who’ve found the cause of their heartburn and indigestion to be low stomach acidity. In nearly all of these folks, symptoms have been relieved andJonathan Wright MD digestion improved when they’ve taken supplemental hydrochloric acid and pepsin capsules. (Certainly it would be preferable that our stomach production of hydrochloric acid and pepsin be restored on its own, but a reliable way to do this hasn’t been found.)”

We are just ‘sheeples’

I’m afraid we are just a load of ‘sheeples’ being led by the nose into a cocophany of illnesses and diseases which are being caused by prescription and over the counter, drugs. But it is unforgivable to convince the general population with heartburn or GERD or other digestive problems, that we have ‘too much acid’, when the majority of us have the exact opposite.  Of course, we could all be in that group of 1 out of 100 that actually do produce too much acid but I don’t think so.  IMHO most of us who suffer ‘acid indigestion’ have brought it on ourselves and we need to take control of our own health and take preventative measures to stop these conditions before they start.

How about a change of lifestyle and a daily dose of Mg Chloride (MgCl2). The clue here is in the ‘chloride’ ie. hydrochloric acid which those with low acid will be deficient in (as well as Mg)!

Ches Power Author

Any comments or experiences regarding the subject of antacids or PPIs would be welcome.  Give us your views and tell us if you have suffered with this complaint and how/if you are now cured.  Good health to you always, Ches


What Is Type 3 Diabetes? – Are you at risk?

What is type 3 diabetes?  We all know there are two main types of diabetes, Type 1 and Type 2.  There is also the transient gestational diabetes which only affects women during pregnancy.  But now diabetes is being linked with Alzheimer’s and it’s being called type 3. Are you at risk? Continue reading What Is Type 3 Diabetes? – Are you at risk?

Talking to a stranger – your doctor

How many of you actually know your doctor?  You may be acquainted with him or her, but they see hundreds of patients so they are not likely to know you very well nor you them. According to a recent report from the Health Foundation, the UK has the fewest doctors per 1,000 patients in the whole of the EU. Continue reading Talking to a stranger – your doctor